What Leads To Rising Cholesterol In The Body

There isn't any clear scientifically proven explanation as to exactly how a diet high in saturated fat could be the cause of rising cholesterol levels in the blood. We also do not know exactly how cholesterol could develop heart disease. There is however a very plausible theory which has a large following in the scientific community.

The LDL connection

It starts with a high level of LDL cholesterol. Excess levels of LDL cholesterol usually result in some of the excess cholesterol to move to the artery walls. The more the LDL levels, the more cholesterol moves out of the bloodstream and into the blood vessel walls. Rigidity of the artery walls may determine how much cholesterol might stay on the artery walls. Smoking, high blood pressure levels, diabetes, as well as other influences such as stress may constrict artery walls. This could result in a disruption in the flow of blood as well as pressure. When this occurs, artery walls may weaken or become scarred in the first layer of the lining allowing LDL to further embed into the artery walls.

The role of swelling

This sets off a sequence of events where the body effectively sabotages itself. Swelling flares up in the affected region causing White blood cells or macrophages to rush to the scene. The macrophages absorb the cholesterol and get engorged further blocking the bloodstream. These cells continue to call for even more reinforcements causing far more congestion for the flow of blood. The macrophages are created to destroy infectious microbes and then vanish are fighting Lipids which by their very nature are being constantly reproduced. A never ending battle rages. This leads to a stable and continuing state of inflammation inside the artery wall. Gradually the overloaded macrophage is killed and all the cholesterol and inflammatory substances are discharged into the artery wall.

Eventually the body forms a cap of sorts over the inflamed wall section. This cap forms plaque that starts the state of atherosclerosis as well as the shrinking of the artery opening and resultant constriction in the blood circulation. If this occur in an artery that leads to the heart, it may impede the flow of blood to the heart. This restriction however does not usually cause a cardiac arrest. Plaque deposits are full of inflammation and LDL. Immune cells, T-lymphocytes and macrophages, are the most severe in terms of containing inflammatory cells. If the plaque deposit has a thin cap, it is more certain to rupture.

Ruptures are the common cause of cardiac arrest.

Should a plaque cap rupture, blood will seep into the artery wall. The normal wound response mechanism starts and clotting agents are sent to the region. Platelets enter the wound to create a scab that inside an artery wall is life threatening. This scab or clot inside an artery is known as thrombus. It could block blood circulation and oxygen to the heart muscle. The section of the heart which is lacking oxygen rich blood begins to die. This is the way cardiac arrest occur. The medical term is known as a myocardial infarction.

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